Epilepsy is one of the dangerous diseases of all time. For centuries, epilepsy has been well known to exist with a superlative power to destroy human health. But till now, no potential remedies for this terrible giant have been innovated. A group of scientists has recently revealed some relative cause of this complex neurological disorder which has already hit roughly 50 million people all over the world. This study has been considered by many to open up a new path in terms of suitable surgery for the epileptic seizures.

According to Science Daily, the team of researchers belonging from Germany claimed that epilepsy actually roots from brain's large-scale epileptic network. Thus, being a "network disease," it comprises throughout brain's complex wiring. The epileptic seizures actually attribute all the active regions of the brain during the seizure attacks and even during the seizure-free timelines. The team further hints that one typical way to get closer to human brain's wiring based complications is by combining the concepts of a timed-based synchronization theory and space-based network theory to construct functional brain networks.

As per a report by Phys.org, the "seizure-generating area" of the brain of epilepsy affected human is the most critical part of it. As Professor Klaus Lehnertz, head of the Neurophysics Group in the Department of Epileptology at the University of Bonn noted "New developments in network theory are providing powerful tools to construct so-called 'functional networks' from observations of brain activities such as the electroencephalogram (EEG), and helping to identify the important nodes and links within such networks."

Lehnertz along with his team of researchers found a link between the network nodes by examining the synchronization based sensations between neuronal signals generated from all existing pairs of network nodes. They indicated those links to be most crucial in terms of epileptic seizures. The study was also officially published on this week's issue of 'Chaos' from AIP Publishing.